Hydroxyurea

證據等級: L5 預測適應症: 10

目錄

  1. Hydroxyurea
  2. Hydroxyurea: From Undocumented Original Indication to Female Breast Carcinoma
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. US Market Information
    7. Cytotoxicity
    8. Safety Considerations
    9. Conclusion and Next Steps
    10. Disclaimer

## 藥師評估報告

Hydroxyurea: From Undocumented Original Indication to Female Breast Carcinoma

One-Sentence Summary

Hydroxyurea (DrugBank DB01005) is a ribonucleotide reductase inhibitor and broad-spectrum cytotoxic/antimetabolite agent; the evidence pack does not record its original approved indication or formal DrugBank MOA text (data gaps). The TxGNN model predicts it may be effective for Female Breast Carcinoma, supported currently by 0 registered clinical trials and 20 publications, most of which are preclinical or early-phase combination-regimen studies.


Quick Overview

Item Content
Original Indication Not documented in evidence pack (data gap)
Predicted New Indication Female Breast Carcinoma
TxGNN Prediction Score 99.97%
Evidence Level L3
US Market Status Not Marketed (未上市)
Number of NDAs 0
Recommended Decision Research Question

Why is This Prediction Reasonable?

Currently, formal DrugBank-style mechanism-of-action text and the drug’s original approved indication are not available in this evidence pack (flagged as data gaps DG001/DG002). Based on the literature collected for this candidate, hydroxyurea is a ribonucleotide reductase (RNR) inhibitor — a broad-spectrum cytotoxic/antimetabolite agent that has historically been used as a component of combination chemotherapy regimens across multiple solid tumors, including breast cancer (e.g., PMID 26844848 describes it as “an antineoplastic drug used for the treatment of leukemia, sickle-cell disease, HIV, psoriasis, thrombocythemia, and various neoplastic diseases”).

Because the evidence pack contains no record of hydroxyurea’s original indication, the relationship between “original use” and “predicted new indication” cannot be characterized directly. What can be assessed is the mechanistic plausibility: RNR inhibition blocks DNA synthesis in rapidly proliferating cells, a mechanism not tissue-specific and therefore theoretically applicable to breast tumor cells as it is to other malignancies.

The literature evidence in this pack largely supports this at a mechanistic/exploratory level — preclinical studies on HU-lipid conjugates, RNR-inhibitor DNA-repair sensitization, and early-phase (Phase I/Phase I-II) combination regimens including hydroxyurea in breast cancer — but there is no modern, breast-cancer-specific randomized controlled trial. The mechanism is reasonable, but specificity and confirmatory clinical evidence for this indication are currently insufficient.


Clinical Trial Evidence

Currently no related clinical trials registered


Literature Evidence

PMID Year Type Journal Key Findings
7914447 1994 Phase I/II trial Bone Marrow Transplantation High-dose cyclophosphamide + thiotepa + hydroxyurea with autologous stem cell rescue as consolidation chemotherapy in 26 women with responding metastatic breast cancer
1957839 1991 Phase I trial American Journal of Clinical Oncology Sequential 5-FU/leucovorin followed by hydroxyurea with allopurinol protection (HALF regimen) in 20 patients with advanced GI and breast cancer
33631478 2021 Review Pathology, Research and Practice Review of long non-coding RNAs in breast cancer pathogenesis, prognosis, and clinical course (contextual, not HU-specific)
38211596 2024 Preclinical Drug Research In-silico design of novel hydroxyurea-lipid drug conjugates targeting the PI3K/AKT/mTOR pathway to improve HU lipophilicity and reduce toxicity in breast cancer therapy
37777742 2023 Preclinical Molecular Cancer Characterizes EYA4’s role in breast cancer progression and metastasis via replication stress avoidance
30692636 2019 Preclinical Oncogene Nucleostemin’s role in genome maintenance and mammary tumor progression; DNA damage susceptibility in tumor cells
32795962 2020 Preclinical DNA Repair 2-hexyl-4-pentynoic acid sensitizes breast tumor cells to hydroxyurea via RPA2 hyperphosphorylation-mediated DNA repair modulation
26844848 2016 Preclinical Cancer Biotherapy & Radiopharmaceuticals Radiolabeling and evaluation of [99mTc(CO)3]+-hydroxyurea and FITC-hydroxyurea as imaging/tracking agents
25814515 2015 Preclinical Molecular Pharmacology Novel RNR inhibitor COH29 inhibits DNA repair in vitro; hydroxyurea referenced as an established clinical RNR-targeting agent, tested in BRCA1-defective breast cancer cells
27504932 2017 Preclinical Journal of Cellular Physiology Chemoresistance characterization of lung (H460) and breast (MCF-7) cancer cells under prolonged serum starvation

US Market Information

Hydroxyurea currently has no marketing authorization on record in this evidence pack’s regulatory dataset (0 licenses; market status: Not Marketed).


Cytotoxicity

Item Content
Cytotoxicity Classification Conventional cytotoxic (ribonucleotide reductase inhibitor / antimetabolite class)
Myelosuppression Risk Please refer to the package insert warnings and precautions
Emetogenicity Classification Please refer to the package insert warnings and precautions
Monitoring Items Please refer to the package insert warnings and precautions
Handling Protection Please refer to the package insert warnings and precautions

Safety Considerations

Please refer to the package insert for safety information.


Conclusion and Next Steps

Decision: Research Question

Rationale: Evidence for the breast carcinoma indication is Level L3 — literature is dominated by preclinical mechanistic studies and small Phase I/I-II combination-regimen trials from the 1990s, with no registered clinical trials and no modern controlled study specific to breast cancer. A blocking data gap (TFDA label/warnings, DG001) also prevents safety evaluation.

To proceed, the following is needed:

  • TFDA package insert (warnings, contraindications) to unblock safety evaluation (DG001)
  • Formal MOA documentation via DrugBank API (DG002)
  • Confirmation of hydroxyurea’s original approved indication(s), currently undocumented in this evidence pack
  • A modern, breast-cancer-specific controlled trial to validate the mechanistic hypothesis before advancing beyond the research-question stage

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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