Larotrectinib

證據等級: L5 預測適應症: 2

目錄

  1. Larotrectinib
  2. Larotrectinib: A Predicted New Indication — Multiple Endocrine Neoplasia
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. Cytotoxicity
    7. Safety Considerations
    8. Conclusion and Next Steps
    9. Disclaimer

## 藥師評估報告

Larotrectinib: A Predicted New Indication — Multiple Endocrine Neoplasia

One-Sentence Summary

Larotrectinib is not currently marketed in Taiwan, and no original-indication data is on file for this evidence pack. The TxGNN model predicts it may be effective for Multiple Endocrine Neoplasia (MEN), but this direction is currently supported by only 1 clinical trial (a broad genomic basket trial, not MEN-specific) and 2 review-type publications, none of which directly test larotrectinib in MEN patients.


Quick Overview

Item Content
Original Indication Not available (no original indication or Taiwan license data on file)
Predicted New Indication Multiple Endocrine Neoplasia
TxGNN Prediction Score 99.24%
Evidence Level L4
US Market Status ✗ Not Marketed (Taiwan)
Number of NDAs 0
Recommended Decision Hold

Why is This Prediction Reasonable?

Detailed original-indication and formal mechanism-of-action records are not available in this evidence pack (DrugBank MOA lookup flagged as a data gap, severity High). Based on the mechanistic notes attached to this prediction, larotrectinib is known to be a highly selective pan-TRK (NTRK1/2/3) kinase inhibitor.

The predicted new indication, Multiple Endocrine Neoplasia (MEN, including MEN2A/2B), is driven primarily by germline RET mutations — a different oncogenic driver than the NTRK fusions larotrectinib targets. The supporting literature actually discusses RET-directed kinase inhibitors (selpercatinib, pralsetinib) and general thyroid-cancer kinase-inhibitor therapy, not larotrectinib itself. The mechanistic overlap is therefore indirect: it would only be relevant in the rare subset of endocrine tumors that happen to carry an NTRK fusion rather than a RET alteration, which the current evidence does not confirm.

A second, lower-ranked candidate from the same model run — HER2-positive breast carcinoma (TxGNN score 99.14%) — shows a similarly indirect link: the supporting study used entrectinib (a different pan-TRK inhibitor) combined with a JAK2 inhibitor in preclinical models, not larotrectinib. Both predictions are best characterized as plausible drug-class hypotheses rather than drug-specific findings at this stage.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT02465060 Phase 2 Active, not recruiting 6,452 MATCH is a genomic-marker-directed basket trial across refractory advanced solid tumors, lymphomas, and myelomas. It includes an NTRK-fusion-positive treatment arm, but is not designed for MEN or for a larotrectinib+MEN combination — relevance graded C (architectural co-occurrence only, not direct causal evidence).

Literature Evidence

PMID Year Type Journal Key Findings
31322645 2019 Review Endocrine Reviews Reviews kinase-inhibitor therapy (vandetanib, cabozantinib, sorafenib, lenvatinib, and mutation-specific agents) for advanced thyroid cancer; general oncology-kinase-inhibitor context, not larotrectinib-specific.
38438731 2024 Review NPJ Precision Oncology Discusses acquired resistance to RET-targeted TKIs (selpercatinib, pralsetinib) in RET-driven medullary thyroid carcinoma; does not address larotrectinib or MEN directly.

Cytotoxicity

Item Content
Cytotoxicity Classification Targeted therapy (selective pan-TRK/NTRK1-3 kinase inhibitor) — not a conventional cytotoxic agent
Myelosuppression Risk Please refer to the package insert warnings and precautions
Emetogenicity Classification Please refer to the package insert warnings and precautions
Monitoring Items Please refer to the package insert warnings and precautions
Handling Protection Please refer to the package insert warnings and precautions

Safety Considerations

Please refer to the package insert for safety information.


Conclusion and Next Steps

Decision: Hold

Rationale:

  • The mechanistic link between larotrectinib (pan-TRK inhibitor) and MEN (predominantly RET-driven) is indirect, and the sole clinical trial is a non-indication-specific basket trial with only C-grade relevance. Evidence level is L4 (mechanism/preclinical-level), and the model’s own recommendation for this candidate is already “Hold.”
  • TFDA labeling data (Blocking gap) is missing, which prevents even an initial S1 safety screen from starting.

To proceed, the following is needed:

  • TFDA package insert / label data (warnings, contraindications) — currently blocking
  • Confirmed DrugBank mechanism-of-action record
  • MEN-subgroup or NTRK-fusion-stratified outcome data from the MATCH trial (or a dedicated larotrectinib+MEN study)
  • Confirmation of larotrectinib’s original approved indication(s) and Taiwan regulatory/market status, since none is currently on file

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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