Nitroglycerin

證據等級: L5 預測適應症: 5

目錄

  1. Nitroglycerin
  2. Nitroglycerin: From Angina Pectoris to Pulmonary Hypertension
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. US Market Information
    7. Safety Considerations
    8. Conclusion and Next Steps
    9. Disclaimer

## 藥師評估報告

Nitroglycerin: From Angina Pectoris to Pulmonary Hypertension

One-Sentence Summary

Nitroglycerin is a classic nitrate vasodilator historically used for angina pectoris and acute cardiovascular vasospastic conditions. The TxGNN model predicts it may be effective for Pulmonary Hypertension, with 13 clinical trials and 20 publications currently supporting this direction.


Quick Overview

Item Content
Original Indication Angina pectoris / acute vasodilator therapy (regulatory license text not available in this evidence pack)
Predicted New Indication Pulmonary Hypertension
TxGNN Prediction Score 99.61%
Evidence Level L2
US Market Status Not Marketed
Number of NDAs 0
Recommended Decision Proceed with Guardrails

Why is This Prediction Reasonable?

Detailed structured MOA data for nitroglycerin is not available in this evidence pack. Based on the mechanistic rationale generated alongside this prediction, nitroglycerin is metabolized within vascular smooth muscle cells to release nitric oxide (NO), which activates guanylate cyclase and raises intracellular cGMP, producing smooth muscle relaxation. In the pulmonary vascular bed, this pathway can trigger selective pulmonary vasodilation — the same NO/cGMP axis exploited by established pulmonary hypertension therapies such as inhaled nitric oxide (iNO) and PDE5 inhibitors (e.g., sildenafil).

Nitroglycerin’s well-established original use as a systemic vasodilator in angina pectoris and acute coronary vasospasm shares direct mechanistic overlap with pulmonary vasodilation: both indications rely on nitrate-induced smooth muscle relaxation to relieve vascular resistance and improve perfusion. This shared pathway is why nitroglycerin has already been used off-label in acute settings — as a vasoreactivity testing agent in pulmonary arterial hypertension, and as a nebulized adjunct in persistent pulmonary hypertension of the newborn (PPHN) — providing real-world clinical precedent that reinforces the plausibility of the TxGNN prediction.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT01120964 Phase 1/2 Completed 22 RCT of IV L-citrulline (NO precursor pathway) vs placebo in children with pulmonary hypertension after cardiopulmonary bypass
NCT07214129 N/A Completed 20 Nebulized nitroglycerin evaluated directly as a vaso-reactivity testing agent in pulmonary arterial hypertension
NCT05741229 N/A Completed 80 Nebulized nitroglycerin as adjuvant therapy for persistent pulmonary hypertension of the newborn (PPHN); echocardiographic and clinical endpoints
NCT04594629 Phase 1 Unknown 120 Nebulized nitroglycerin vs nebulized PGI2 (epoprostenol) for pulmonary hypertension after valve replacement surgery
NCT00449059 Phase 4 Completed 20 Acute IV nitroglycerin infusion effect on cyclosporine-induced hypertension after cardiac transplantation
NCT03259165 Phase 2 Terminated 52 Nitroglycerin vs furosemide, lung ultrasound-guided pilot trial in acute heart failure/pulmonary congestion
NCT06107465 Phase 2/3 Unknown 60 High- vs low-dose nitroglycerin in sympathetic crashing acute pulmonary edema
NCT02966665 Phase 1 Recruiting 420 Vascular endothelial function and vasomotor tone study in hypertension (indirect relevance)
NCT05172739 Phase 4 Recruiting 70 Perioperative opioid-free anesthesia strategy in lung cancer surgery (nitroglycerin not primary intervention)
NCT05373108 Phase 4 Completed 19 Endothelin-1 and vasomotor function in cardiac allograft vasculopathy after heart transplant

Literature Evidence

PMID Year Type Journal Key Findings
40888971 2025 RCT European Journal of Pediatrics RCT (n=80) showing nebulized nitroglycerin improves echocardiographic/clinical parameters in persistent pulmonary hypertension of the newborn
29880427 2018 RCT (Cohort) J Cardiothorac Vasc Anesth Randomized study: dobutamine + nitroglycerin vs milrinone for perioperative pulmonary hypertension in mitral valve surgery
6423015 1984 Clinical study Bull Eur Physiopathol Respir Sublingual nitroglycerin reduced pulmonary arterial pressure and pulmonary vascular resistance in COPD-related pulmonary hypertension
34082850 2021 Cohort/Case series Cardiology in the Young Review/case series on nitroglycerin inhalation for acute pulmonary arterial hypertension in children with congenital heart disease
6407380 1983 Clinical study Annals of Internal Medicine Nitroglycerin increased cardiac index, decreased pulmonary vascular resistance and mean pulmonary artery pressure in 9 patients with chronic pulmonary hypertension
15947535 2005 Retrospective study Congestive Heart Failure Acute hemodynamic effects of IV nitroglycerin vs epoprostenol in pulmonary arterial hypertension (n=59)
14508317 2003 Clinical study Anesthesiology Nitroglycerin inhalation improved postoperative hemodynamics in pulmonary hypertension patients undergoing mitral valve replacement
10214095 1999 Case series West Virginia Medical Journal Nebulized nitroglycerin treated pulmonary hypertension in children with congenital heart disease
3096761 1986 Clinical study Eur J Respir Dis Suppl Transdermal nitroglycerin patches produced sustained reduction in pulmonary artery pressure and vascular resistance over 4 weeks
29096811 2017 Review J Am Coll Cardiol Comprehensive review of nitroglycerin/nitrogen oxide pharmacology and vasodilatory mechanism relevant to pulmonary and systemic circulation

US Market Information

Nitroglycerin is not currently marketed under the jurisdiction covered by this evidence pack (market status: Not Marketed, 0 licenses on file). No authorization records are available for review.


Safety Considerations

Please refer to the package insert for safety information.


Conclusion and Next Steps

Decision: Proceed with Guardrails

Rationale: Evidence level L2 is supported by a 2025 RCT in neonatal PPHN, several controlled/randomized perioperative studies, and decades of consistent hemodynamic data showing nitroglycerin reduces pulmonary artery pressure and pulmonary vascular resistance. However, most supportive studies are small, single-center, or focused on acute/perioperative settings rather than chronic pulmonary arterial hypertension, so confirmatory large-scale Phase 3 trials are still lacking.

To proceed, the following is needed:

  • TFDA/regulatory label warnings and contraindications (currently a Blocking data gap — required before any safety pre-assessment)
  • Structured mechanism of action (MOA) data from DrugBank (High-priority gap)
  • Drug-drug interaction (DDI) profile — current query returned no results
  • A dedicated safety monitoring plan for hemodynamic effects (hypotension, reflex tachycardia, nitrate tolerance) if pulmonary hypertension use is pursued
  • Consideration of confirmatory controlled trials specifically in chronic pulmonary arterial hypertension populations

Note: Prinzmetal angina (rank 3, L2, Proceed with Guardrails) shows comparably strong mechanistic and historical clinical evidence and may warrant a parallel evaluation. Kyphoscoliotic heart disease, primary hereditary glaucoma, and congenital hypotrichosis milia (all L5, Hold) currently lack supporting clinical or literature evidence and are not recommended for further investment at this time.

Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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