Vinblastine

證據等級: L5 預測適應症: 10

目錄

  1. Vinblastine
  2. Vinblastine: From Cytotoxic Chemotherapy to Rhabdomyosarcoma
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. US Market Information
    7. Cytotoxicity
    8. Safety Considerations
    9. Conclusion and Next Steps
    10. Disclaimer

## 藥師評估報告

Vinblastine: From Cytotoxic Chemotherapy to Rhabdomyosarcoma

One-Sentence Summary

Vinblastine is a vinca alkaloid antineoplastic agent whose specific original indication is not documented in this evidence pack (the drug is not currently marketed in the reviewed jurisdiction). The TxGNN model predicts it may be effective for Rhabdomyosarcoma, supported by 0 registered clinical trials and 16 relevant publications, including case reports, pilot cohorts, and one Phase 2 RCT of a related vinca alkaloid (vinorelbine).


Quick Overview

Item Content
Original Indication Not specified in evidence pack; vinblastine is a historically established cytotoxic antineoplastic agent (vinca alkaloid class)
Predicted New Indication Rhabdomyosarcoma
TxGNN Prediction Score 99.86%
Evidence Level L3
US Market Status Not Marketed
Number of NDAs 0
Recommended Decision Hold

Why is This Prediction Reasonable?

Currently, detailed mechanism of action data is not available in the evidence pack (original_moa: Data Gap). Based on the known pharmacology captured in the repurposing rationale, vinblastine is a vinca alkaloid that inhibits tubulin/microtubule polymerization, thereby blocking mitosis. This is the same mechanistic class as vincristine and vinorelbine, both of which are established components of standard rhabdomyosarcoma chemotherapy regimens.

Rhabdomyosarcoma is a highly proliferative pediatric/adult soft-tissue sarcoma, and microtubule-targeting agents are a core pillar of its treatment. The literature evidence directly supports this mechanistic link: a preclinical xenograft study (PMID 3329524) found vinblastine and vincristine to be the only vinca alkaloids with demonstrated clinical utility against human rhabdomyosarcoma, and multiple case reports describe vinblastine used within combination regimens (e.g., cisplatin-vinblastine-bleomycin, PVP therapy) for prostate and perianal rhabdomyosarcoma. While vinblastine itself has not been tested in a dedicated large-scale trial for this indication, its structural analogue vinorelbine has Phase 2 RCT-level evidence of activity in relapsed/refractory rhabdomyosarcoma (PMID 22633624), reinforcing the plausibility of the class effect.


Clinical Trial Evidence

Currently no related clinical trials registered


Literature Evidence

PMID Year Type Journal Key Findings
22633624 2012 RCT (Phase 2) European Journal of Cancer Vinorelbine + low-dose cyclophosphamide showed good tolerance and efficacy in relapsed/refractory pediatric solid tumours, with activity specifically noted in rhabdomyosarcoma
22156656 2011 Cohort (Pilot) Oncotarget Pilot study of a 4-drug pediatric metronomic regimen for resistant cancers
15378498 2004 Cohort (Pilot) Cancer Vinorelbine + low-dose cyclophosphamide pilot study to define optimal dosing ahead of European Rhabdomyosarcoma Protocol
12115359 2002 Cohort Cancer Vinorelbine showed evidence of activity in previously treated advanced childhood rhabdomyosarcoma
38050209 2023 Case Report/Review Medicine Adult perianal rhabdomyosarcoma with nodal metastases achieved partial response after nivolumab, dacarbazine, cisplatin, and vinblastine combination therapy
3329524 1987 Mechanistic (Preclinical) Anti-Cancer Drug Design Xenograft model of human rhabdomyosarcoma identified vincristine and vinblastine as the only vinca alkaloids with demonstrated clinical utility against this tumour type
2451411 1987 Case Report Hinyokika Kiyo Refractory prostatic rhabdomyosarcoma in a child responded to combination cisplatin, vinblastine, and peplomycin (PVP) therapy after relapse
26024389 2015 Preclinical Cell Death and Differentiation Identified synthetic lethality between PLK1 inhibitors and microtubule-destabilizing drugs (vinblastine’s mechanistic class) in preclinical rhabdomyosarcoma models
2412542 1985 Case Report ANZ Journal of Surgery Rhabdomyosarcoma developed in a patient previously treated with chemotherapy for metastatic germ cell testicular tumour
41216926 2026 Prospective Trial (pending classification) Pediatric Blood & Cancer CWS-96/CWS-2002P prospective European trials evaluating risk stratification and chemotherapy regimens across soft tissue sarcoma entities, reclassified against a recent registry

US Market Information

Vinblastine is currently not marketed in the reviewed jurisdiction (market status: Not Marketed), and no license/NDA records are available in the evidence pack (total_licenses: 0).


Cytotoxicity

Item Content
Cytotoxicity Classification Conventional cytotoxic (Vinca alkaloid, microtubule polymerization inhibitor)
Myelosuppression Risk Please refer to the package insert warnings and precautions
Emetogenicity Classification Please refer to the package insert warnings and precautions
Monitoring Items Please refer to the package insert warnings and precautions
Handling Protection As a conventional cytotoxic agent (vinca alkaloid class), standard cytotoxic drug handling precautions should apply

Safety Considerations

Please refer to the package insert for safety information.


Conclusion and Next Steps

Decision: Hold

Rationale: Mechanistic plausibility is strong (vinca alkaloid class effect shared with vincristine/vinorelbine, both standard-of-care in rhabdomyosarcoma), and it is corroborated by one Phase 2 RCT of a related agent plus multiple case reports directly using vinblastine in rhabdomyosarcoma regimens. However, there are no registered clinical trials of vinblastine itself for this indication, and a Blocking-severity data gap exists for prescribing/safety information (TFDA-equivalent warnings and contraindications), which prevents a safety pre-assessment (S1) from being completed.

To proceed, the following is needed:

  • Package insert warnings, contraindications, and DDI data (currently Blocking data gap, DG001)
  • Confirmed mechanism of action documentation (DG002)
  • A dedicated clinical trial or larger case series evaluating vinblastine specifically (not only related vinca alkaloids) in rhabdomyosarcoma
  • Myelosuppression/emetogenicity and monitoring protocol specific to the intended dosing regimen

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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